Surveillance的問題，透過圖書和論文來找解法和答案更準確安心。 我們查出實價登入價格、格局平面圖和買賣資訊

Worm: A Cuban American Odyssey

為了解決Surveillance的問題，作者Rodriguez, Edel 這樣論述：

From ＂America’s illustrator in chief＂ (Fast Company), a stunning graphic memoir of a childhood in Cuba, coming to America on the Mariel boatlift, and a defense of democracy, here and thereHailed for his iconic art on the cover of Time and on jumbotrons around the world, Edel Rodriguez is among th

e most prominent political artists of our age. Now for the first time, he draws his own life, revisiting his childhood in Cuba and his family’s passage on the infamous Mariel boatlift. When Edel was nine, Fidel Castro announced his surprising decision to let 125,000 traitors of the revolution, or ＂w

orms,＂ leave the country. The faltering economy and Edel’s family’s vocal discomfort with government surveillance had made their daily lives on a farm outside Havana precarious, and they secretly planned to leave. But before that happened, a dozen soldiers confiscated their home and property and imp

risoned them in a detention center near the port of Mariel, where they were held with dissidents and criminals before being marched to a flotilla that miraculously deposited them, overnight, in Florida. Through vivid, stirring art, Worm tells a story of a boyhood in the midst of the Cold War, a fami

ly’s displacement in exile, and their tenacious longing for those they left behind. It also recounts the coming-of-age of an artist and activist, who, witnessing American’s turn from democracy to extremism, struggles to differentiate his adoptive country from the dictatorship he fled. Confronting qu

estions of patriotism and the liminal nature of belonging, Edel Rodriguez ultimately celebrates the immigrants, maligned and overlooked, who guard and invigorate American freedom.

Surveillance進入發燒排行的影片

基於積分同步學習之多軸無人機參數估測與控制

為了解決Surveillance的問題，作者楊承澄 這樣論述：

在本篇論文中，基於積分同步學習 (ICL) 之多軸無人機參數估測與控制演算法被完整開發，此控制演算法將被用來同時控制無人機與估測未知的無人機質量與轉動慣量。據我們所知，這是首個將同步學習應用於估測無人機之質量與轉動慣量上的研究，此控制演算法搭配幾何追蹤控制提供無人機迴授控制。在此研究中，由於無人機的動態是在全域的空間上定義，因此開發出來的 ICL 控制器可以確保全域的狀態誤差收斂與參數估測誤差收斂。此外，此控制架構可以被應用在任何未知質量與轉動慣量的無人機上。經由穩定性證明，狀態誤差與參數估測誤差會以漸近的形式收斂。ICL 控制器的性能與效率將會經由模擬與實驗結果展現。

Stay This Day and Night with Me

為了解決Surveillance的問題，作者 這樣論述：

This is the story of Olga, a retired mathematician, and Mateo, a college student passionate about robotics, and their plot to influence Google.After a chance encounter at the public library, two new friends begin to meet up regularly. Together they decide to submit an extremely unorthodox applica

tion to Google’s Singularity University, framed as a direct appeal to the seemingly all-powerful corporation. Ideas and counterarguments open into personal stories as they debate the possibility of free will, the existence of merit, and the role of synthetic humans.As Olga and Mateo craft their conf

rontation with Google, they ask the most basic and important of questions: What does it mean to be human in a world driven by data and surveillance? Is there still space for empathy and caring? What could we be, what could we build, right now, if we used our resources in different ways? Tension grow

s as their relationship becomes strained due to ideological differences, and as Olga confronts a terminal illness, Mateo begins to plan an attack on Google.Author Belén Gopegui has been compared to Cervantes, Nabokov, and Borges. Here, she offers a literary equivalent of My Dinner with Andre, creati

ng a hermetic, compelling world within an impassioned conversation that opens into a rich examination of the rights, roles, and obligations of humans in a world made by algorithms.

潛藏危機：Musashi-1固有無序區域介導與神經退行性疾病相關蛋白之異常聚集

為了解決Surveillance的問題，作者杜岱芸 這樣論述：

蛋白質病變(proteopathy)是退行性疾病的常見原因，通過錯誤折疊的蛋白質異常聚集形成類澱粉沉積症(amyloidogenesis)，從而導致破壞組織內的穩態。尤其是，近期研究表明細胞內具有固有無序區域 (intrinsically disordered regions)的蛋白容易進行液-液相分離(liquid-liquid phase separation)，從而在細胞中組裝蛋白質凝聚層(coacervates)。在本研究中，我們假設具有固有無序區域的蛋白質受環境壓力影響，促進異常折疊甚至形成聚集體，這將進一步形成澱粉樣斑塊(amyloid plaques)並在組織內堆積，導致蛋白質

病變。我們主要探討不僅是RNA結合蛋白、也是幹性基因的Musashi-1，是否與具有豐富IDR的Musashi-1 C-末端區域相互作用以進行液-液相分離，最終形成澱粉樣原纖維(amyloid fibrils)。為了確認哪些序列更易於形成澱粉樣蛋白，因此對Musashi-1的C-末端進行了序列連續刪除來取得不同長度的片段。我們的研究結果表明Musashi-1 C-末端面對不同pH值和鹽濃度會影響液-液相分離狀態，包含改變蛋白質相分離的出現時間、形狀和大小，隨著時間的推移，Musashi-1 C-末端也可以形成澱粉樣蛋白原纖維。而當在氧化壓力下，它會在細胞內誘導組裝應激顆粒與不可逆的聚集體的形成

，另一方面，當細胞同時表達Musashi-1 C-末端和內源性TDP-43，Musashi-1 C-末端誘導TDP-43從細胞核錯誤定位到細胞質。此外，Musashi-1 C-末端促進磷酸化和泛素化TDP-43。總結來說，我們提出了關於Musashi-1與神經退行性疾病相關蛋白相互作用導致異常聚集的新見解，這些發現有助於提供解決退行性疾病的新思路。